A new study has found elderly individuals with low Vitamin A levels may be more at risk of developing Alzheimer’s disease along with lower brain functioning. Additionally the study found the even a marginal Vitamin A deficiency at birth might affect long-term risk factors.
This new study was made up of findings from mouse models as well as human population data. The population study revealed that 75% of those with either a significant or even mild Vitamin A deficiency had some cognitive impairment compared to 47% with normal Vitamin A levels that experienced cognitive impairment. The research on mice confirmed this finding. Even a mild Vitamin A deficiency during pregnancy or immediately after birth was associated with an increase in the production of amyloid-beta plaques which are associated with Alzheimer’s disease. Researchers found that mice deprived of Vitamin A performed poorly on standardized tests of memory as well as learning as adults. Mice that had Vitamin A withheld while they were in the womb but were given a normal diet after they were born had lower performance in general when compared to mice receiving adequate Vitamin A in the womb but were deprived Vitamin A after birth. It was found that some reversal of decline could be reversed with adequate Vitamin A supplementation. Researchers concluded that monitoring Vitamin A during infancy as well as during pregnancy and eliminating a prenatal Vitamin A deficiency may aid in halting Alzheimer’s disease development.
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